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Immune & Thymic

KPV

Also known as · α-MSH(11-13)

Overview

The C-terminal tripeptide of α-MSH, isolated for its anti-inflammatory activity in models of colitis, dermatitis, and mast-cell-mediated inflammation.

Research profile · for clinicians

Based on research: the C-terminal fragment of α-MSH, isolated specifically for its anti-inflammatory activity. Preclinical models of colitis, mast-cell activation, and inflammatory skin conditions report reduced NF-κB signaling and lower pro-inflammatory cytokine output.

Mechanism of action

Inhibits NF-κB signaling and reduces pro-inflammatory cytokine release (TNF-α, IL-1β) in epithelial and immune cells; melanocortin-receptor-independent intracellular activity has been reported.

Research record

  • [01]Murine models of DSS-induced colitis.
  • [02]Topical and oral preclinical studies of inflammatory skin conditions.

Selected references

  • The C-terminal tripeptide of α-MSH exerts anti-inflammatory actions

    FASEB J., 2002