Tesamorelin
Overview
An N-terminally modified GHRH analogue investigated in HIV-associated lipodystrophy research. The hexenoyl modification confers resistance to dipeptidyl-peptidase IV degradation.
Research profile · for clinicians
Based on research: best characterized in the visceral-fat and metabolic literature. Elevates the body's own GH and downstream IGF-1, which in HIV-associated lipodystrophy trials tracked with meaningful visceral adipose tissue reduction; also explored in fatty-liver cohorts.
Mechanism of action
Stimulates endogenous GH release via GHRH receptor agonism; downstream IGF-1 elevation has been associated with visceral adipose tissue reduction in study cohorts.
Research record
- [01]Reduction of visceral adipose tissue in HIV-lipodystrophy trials.
- [02]Investigations in non-alcoholic fatty liver disease cohorts.
Selected references
Effects of tesamorelin on VAT
JAMA, 2007